Investigating the Molecular Mechanism of Quercetin Protecting against Podocyte Injury to Attenuate Diabetic Nephropathy through Network Pharmacology, MicroarrayData Analysis, and Molecular Docking

<div>(a) 41 candidate targets obtained from network of “protein-protein” interaction (PPI), including three key targets TNF (degree = 34), VEGFA (degree = 33), and AKT1 (degree = 31). The color of the nodes is shown in a gradient from to red to transparent according to the degree value. (b) 13 DEGs contained in 41 candidate targets, the red bars represents11 upregulated genes (log C 1), and the green bars represents 2 upregulated genes (log FC &lt; −1).</div>

Evidence-Based Complementary and Alternative Medicine

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Figure 3: Investigating the Molecular Mechanism of Quercetin Protecting against Podocyte Injury to Attenuate Diabetic Nephropathy through Network Pharmacology, MicroarrayData Analysis, and Molecular Docking 

Figure 3 | Investigating the Molecular Mechanism of Quercetin Protecting against Podocyte Injury to Attenuate Diabetic Nephropathy through Network Pharmacology, MicroarrayData Analysis, and Molecular Docking 