Research Article

Pleckstrin-2 as a Prognostic Factor and Mediator of Gastric Cancer Progression

Figure 4

Overexpression of PLEK2 promoted gastric cancer proliferation, colony formation, and cell migration. (a) The overexpression of endogenous PLEK2 was confirmed at the mRNA and protein levels. (b) The overexpression of PLEK2 increased the cell proliferation rate in MGC803 and SGC7901 cells. (c) The overexpression of PLEK2 enhanced the colony formation ability of gastric cancer cells. (d) The overexpression of PLEK2 accelerated gastric cancer cell migration. (e) The overexpression of PLEK2 enhanced gastric cancer cell wound healing ability. , , and (-test).
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