Study of the Mechanism by Which Curcumin Cooperates with Sestrin2 to Inhibit the Growth of Pancreatic Cancer

Figure 1

Evaluation of the pancreatic cancer cell models modified by sestrin2 overexpression/knockdown lentivirus. The expression of sestrin2 in pancreatic cancer cells infected by sestrin2 overexpression and knockdown lentivirus was visualized by western blotting (a, b), while the β-actin was utilized to be an internal control; the expression of sestrin2 mRNA in pancreatic cancer cells infected by sestrin2 overexpression and knockdown lentivirus was visualized by q-RT PCR (c, d).

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