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International Journal of Alzheimer’s Disease
Volume 2012, Article ID 707482, 2 pages

Tau Protein: Function and Pathology

1Department of Neurology, The Agnes Ginges Center for Human Neurogenetics, Hadassah Hebrew University Medical Center, Ein Karem, Jerusalem 91120, Israel
2Inserm U837, “Alzheimer & Tauopathies” Jean-Pierre Aubert Research Centre, University Lille-Nord de France, UDSL, Lille, France
3Departments of Neurology and Neuroscience & Cell Biology, George and Cynthia Mitchell Center for Neurodegenerative Disease, University of Texas Medical Branch, Galveston, TX 77555, USA
4Alzheimer's and Parkinson's Disease Laboratory, Brain and Mind Research Institute, University of Sydney, Camperdown, NSW 2050, Australia

Received 17 July 2012; Accepted 17 July 2012

Copyright © 2012 Hanna Rosenmann et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Citations to this Article [2 citations]

The following is the list of published articles that have cited the current article.

  • Maria del Carmen Cardenas-Aguayo, Laura Gomez-Virgilio, Steven DeRosa, and Marco Antonio Meraz-Rios, “The Role of Tau Oligomers in the Onset of Alzheimer's Disease Neuropathology,” Acs Chemical Neuroscience, vol. 5, no. 12, pp. 1178–1191, 2014. View at Publisher · View at Google Scholar
  • Nicole Maphis, Shanya Jiang, Guixiang Xu, Olga N. Kokiko-Cochran, Saktimayee M. Roy, Linda J. Van Eldik, D. Martin Watterson, Bruce T. Lamb, and Kiran Bhaskar, “Selective suppression of the α isoform of p38 MAPK rescues late-stage tau pathology,” Alzheimer's Research & Therapy, vol. 8, no. 1, 2016. View at Publisher · View at Google Scholar