The Role of Aryl Hydrocarbon Receptor and Crosstalk with Estrogen Receptor in Response of Breast Cancer Cells to the Novel Antitumor Agents Benzothiazoles and Aminoflavone

<table>DF 203 causes an increase in immunoreactive nuclear AhR in MCF-7 cells. MCF-7 and MDA-MB-435 were treated with 0.1% DMSO, 1 <i>μ</i>M DF 203, or 20 <i>μ</i>M <i>β</i>-naphthoflavone for 1 h. Determination of immunoreactive AhR in cytoplasmic and nuclear fractions was performed by western blot. <i>β</i>-NF: <i>β</i>-naphthoflavone [<a href="/journals/ijbc/2011/923250/#B15">15</a>].</table>

International Journal of Breast Cancer

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Figure 3: The Role of Aryl Hydrocarbon Receptor and Crosstalk with Estrogen Receptor in Response of Breast Cancer Cells to the Novel Antitumor Agents Benzothiazoles and Aminoflavone 

Figure 3 | The Role of Aryl Hydrocarbon Receptor and Crosstalk with Estrogen Receptor in Response of Breast Cancer Cells to the Novel Antitumor Agents Benzothiazoles and Aminoflavone 