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International Journal of Endocrinology
Volume 2013, Article ID 259189, 8 pages
http://dx.doi.org/10.1155/2013/259189
Research Article

Transient Neonatal Zinc Deficiency Caused by a Heterozygous G87R Mutation in the Zinc Transporter ZnT-2 (SLC30A2) Gene in the Mother Highlighting the Importance of Zn2+ for Normal Growth and Development

1Division of Paediatric Endocrinology, Diabetology and Metabolism and Department of Clinical Research, University Children’s Hospital, Inselspital, 3010 Bern, Switzerland
2Department of Dermatology, University of Bern, 3010 Bern, Switzerland

Received 12 June 2013; Revised 7 August 2013; Accepted 22 August 2013

Academic Editor: Fabio Buzi

Copyright © 2013 Maria Consolata Miletta et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

How to Cite this Article

Maria Consolata Miletta, Andreas Bieri, Kristin Kernland, et al., “Transient Neonatal Zinc Deficiency Caused by a Heterozygous G87R Mutation in the Zinc Transporter ZnT-2 (SLC30A2) Gene in the Mother Highlighting the Importance of Zn2+ for Normal Growth and Development,” International Journal of Endocrinology, vol. 2013, Article ID 259189, 8 pages, 2013. https://doi.org/10.1155/2013/259189.