TET3 Mediates 5hmC Level and Promotes Tumorigenesis by Activating AMPK Pathway in Papillary Thyroid Cancer

Figure 5

(a) Western blotting was used to detect phosphorylated AMPK, phosphorylated mTOR, phosphorylated ULK1, ATG5, and LC3BII/I in BCPAP and TPC-1 cells treated with TET3 knockdown or AMPK knockdown. CCK-8 assay (b) and colony formation assay (c) showed that AMPK knockdown partially attenuated the inhibited proliferation induced by TET3 knockdown in BCPAP and TPC-1 cells.

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