Correlations between Endothelial Functions and ROS Detection in Diabetic Microvascular Wall: Early and Late Ascorbic Acid Supplementation
Figure 6
The diagram represents the proposed mechanism of ascorbic acid (AA) in attenuating reactive oxygen species (ROS) production when DAHP inhibited BH4 and caused eNOS uncoupling. Similar to diabetic-induced endothelial cells dysfunction in associated with BH4 deficiency.
