Effect of Chronic L-Dopa or Melatonin Treatments after Dopamine Deafferentation in Rats: Dyskinesia, Motor Performance, and Cytological Analysis

<table>Integrated AIM scores were generated separately for locomotive (a), orolingual (b), axial (c), and forelimb (d) AIMs using data from day 30 of chronic treatments. Note that animals treated with melatonin did not develop locomotive AIMs and axial rotation (<sup><i>∗</i></sup><svg height="11.25" id="M9" style="vertical-align:-0.30096pt" version="1.1" viewbox="0 0 63.962502 11.25" width="63.962502" xmlns="http://www.w3.org/2000/svg">
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<tspan style="font-size: 12.50px; " x="0" y="0">𝑃</tspan>
<tspan style="font-size: 12.50px; " x="10.890113" y="0">&lt;</tspan>
<tspan style="font-size: 12.50px; " x="22.930502" y="0">0</tspan>
<tspan style="font-size: 12.50px; " x="29.182001" y="0">.</tspan>
<tspan style="font-size: 12.50px; " x="32.307751" y="0">0</tspan>
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</svg> LD-treated group versus 6-OHDA-lesioned and melatonin-treated groups).</table>

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fig5

Figure 5

Figure 5: Effect of Chronic L-Dopa or Melatonin Treatments after Dopamine Deafferentation in Rats: Dyskinesia, Motor Performance, and Cytological Analysis 

Figure 5 | Effect of Chronic L-Dopa or Melatonin Treatments after Dopamine Deafferentation in Rats: Dyskinesia, Motor Performance, and Cytological Analysis 