Genetic and Epigenetic Tumor Suppressor Gene Silencing Are Distinct Molecular Phenotypes Driven by Growth Promoting Mutations in Nonsmall Cell Lung Cancer

<table>Model of the molecular
phenotypes of NSCLC driven by promoter hypermethylation propensity. The model
depicts the inverse relationship between epigenetic and genetic alterations  and its
positive association between epigenetic propensity and growth promoting
oncogene activation.</table>

Journal of Cancer Epidemiology

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Figure 2

Figure 2: Genetic and Epigenetic Tumor Suppressor Gene Silencing Are Distinct Molecular Phenotypes Driven by Growth Promoting Mutations in Nonsmall Cell Lung Cancer 

Figure 2 | Genetic and Epigenetic Tumor Suppressor Gene Silencing Are Distinct Molecular Phenotypes Driven by Growth Promoting Mutations in Nonsmall Cell Lung Cancer 