Coenzyme Q10 Attenuates High Glucose-Induced Endothelial Progenitor Cell Dysfunction through AMP-Activated Protein Kinase Pathways

Figure 1

Morphology and characterization of EPCs from peripheral blood. (a) Peripheral blood mononuclear cells (MNCs) were plated on a fibronectin-coated culture dish on the fourteenth day. EPCs were also characterized by immunofluorescence staining for the expression of (b) DiI-AcLDL, (c) lectin, (d) CD133, (e) kinase insert domain receptor (KDR), (f) platelet/endothelial cell adhesion molecule-1 (CD31), (g) CD34, (h) VE-cadherin, and (i) Von Willebrand factor (vWF). Cells were counterstained with 4′,6-diamidino-2-phenylindole (DAPI) for the nuclei (blue).

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