Urinary Sodium Concentration Is an Independent Predictor of All-Cause and Cardiovascular Mortality in a Type 2 Diabetes Cohort Population

Table 2

Association of clinical and biological characteristic variables with measured urinary sodium concentration (Spearman’s correlation).

	Estimate (95% CI)	value
Age	−0.09 (−0.15 to −0.04)	<0.001
Male gender	−0.16 (−0.26 to −0.05)	0.004
History of cardiovascular disease	−0.18 (−0.31 to −0.05)	0.008
Use of diuretics	−0.30 (−0.41 to −0.20)	<0.001
Use of insulin	−0.24 (−0.34 to −0.14)	<0.001
eGFR	0.28 (0.23 to 0.33)	<0.001
uACR	−0.14 (−0.19 to −0.09)	<0.001
	0.18 (0.13 to 0.24)	<0.001
U :	0.18 (0.13 to 0.23)	<0.001
estimated 24 h Na excretion	0.29 (0.26 to 0.31)	<0.001
NT-proBNP	−0.22 (−0.27 to −0.17)	<0.001
Glycosuria	0.13 (0.03 to 0.24)	0.02

Estimates are calculated for 1-SD increment of continuous variables.
eGFR, estimated glomerular filtration rate; uACR, urinary albumin/creatinine ratio; , urinary potassium concentration; U : , urine to plasma creatinine ratio; NT-proBNP, N-terminal pro-brain natriuretic peptide.
uACR, copeptin, and NT-proBNP are log-transformed.