Group 2 Innate Lymphoid Cells Participate in Renal Fibrosis in Diabetic Kidney Disease Partly via TGF-β1 Signal Pathway

Table 2

Characteristics of the normal control subjects (NC) or patients with type 2 diabetes (DM), early diabetic kidney disease (DKD1), and late diabetic kidney disease (DKD2) at baseline.

Items	NC	DM	DKD1	DKD2
Gender (male/female)	16/14	16/14	9/6	8/7
Age (year)					1.771	0.156
BMI (kg/m^2)					3.020	0.032
SBP (mmHg)					9.248	<0.01
DBP (mmHg)					1.609	0.190
FBG (mmol/L)					41.083	<0.01
HbA1c (%)					34.566	<0.01
WBC (10^9/L)					19.205	<0.01
Neu (10^9/L)					16.688	<0.01
Lym (10^9/L)					5.594	<0.01
TG (mmol/L)					7.097	<0.01
TC (mmol/L)					2.362	0.074
HDL-C (mmol/L)					10.173	<0.01
LDL-C (mmol/L)					2.553	0.058
Cr (μmol/L)					240.383	<0.01
UA (μmol/L)					11.670	<0.01
UAER (mmol/L)					32.558	<0.01
eGFR (mL/min·1.73 m^2)					108.749	<0.01

BMI: body mass index, SBP: systolic pressure, DBP: diastolic pressure, FBG: fasting blood glucose, HbA1c: glycosylated hemoglobin A1c, WBC: white blood cell, Neu: neutrophil, Lym: lymphocyte, TG: triglyceride, TC: cholesterol, HDL-C: high-density lipoprotein cholesterol, LDL-C: low-density lipoprotein cholesterol, Cr: creatinine, UA: uric acid, eGFR: estimated glomerular filtration rate. The and represent the results of comparison between groups.