Clinical Isolates of <i>Mycobacterium tuberculosis</i> Differ in Their Ability to Induce Respiratory Burst and Apoptosis in Neutrophils as a Possible Mechanism of Immune Escape

<table>Spoligotyping and IS6110-RFLP pattern profiles of <i>Mtb</i> strains used as antigens in this study: reference virulent strain H37Rv, local-drug-sensitive (LAM, 10406) and drug-resistant (Ra, 11608) LAM strains, drug-sensitive Haarlem strain (H, 12425), and MDR Haarlem strain, M (6548).</table>

Journal of Immunology Research

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Figure 1

Figure 1: Clinical Isolates of <i>Mycobacterium tuberculosis</i> Differ in Their Ability to Induce Respiratory Burst and Apoptosis in Neutrophils as a Possible Mechanism of Immune Escape 

Figure 1 | Clinical Isolates of Mycobacterium tuberculosis Differ in Their Ability to Induce Respiratory Burst and Apoptosis in Neutrophils as a Possible Mechanism of Immune Escape 