The Apolipoprotein C-I Content of Very-Low-Density Lipoproteins Is Associated with Fasting Triglycerides, Postprandial Lipemia, and Carotid Atherosclerosis
Figure 3
Line graphs showing serum concentrations of triglycerides (Tg) over time in groups after intake of a standard high-fat meal (a) and changes in area under the curve (AUC) for serum triglycerides in the postprandial state (0–8 hrs) across tertiles of the number of apoC-I per VLDL particle in the postprandial state (b). Groups (T1–T3) were defined by their respective tertiles of the number of apoC-I per VLDL particle in the postprandial state. Tertiles of the number of apoC-I per VLDL particle in the postprandial state were as follows (mean, range): T1 (5.3, 1.1–7.9), T2 (10.5, 8.1–13.0), and T3 (22.6, 13.7–40.3). Values are means ± SEM. Linear trend analyses were adjusted for age, BMI, the content of apoC-III per VLDL particle, and APOE genotyping.
