Ginsenoside Rh2 Inhibits Glycolysis through the STAT3/c-MYC Axis in Non-Small-Cell Lung Cancer

Figure 7

Schematic representation of the antitumor effect of Rh2 in NSCLC cells. Rh2 inhibited NSCLC cell invasion and migration by suppressing EMT and promoted apoptosis through regulating the mitochondrial apoptotic way. The metabolic shift induced by Rh2 in NSCLC depended on regulating the expression of glycolysis-related enzymes via the STAT3/c-Myc axis.