Regulation of the Inflammatory Response, Proliferation, Migration, and Epithelial-Mesenchymal Transition of Human Lens Epithelial Cells by the lncRNA-MALAT1/miR-26a-5p/TET1 Signaling Axis

<div>TNF-<i>α</i> treatment induces inflammatory injured HLECs. (a) Morphological changes of HLECs after TNF-<i>α</i> addition (scale: 200 <i>μ</i>m). Inflammatory HLECs were transformed into long spindle cells (arrows). (b, c) Evaluation of the changes in inflammatory factor and MALAT1 expressions by RT-qPCR in TNF-<i>α</i> treated HLECs.</div>

Journal of Ophthalmology

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Figure 3

Figure 3: Regulation of the Inflammatory Response, Proliferation, Migration, and Epithelial-Mesenchymal Transition of Human Lens Epithelial Cells by the lncRNA-MALAT1/miR-26a-5p/TET1 Signaling Axis 

Figure 3 | Regulation of the Inflammatory Response, Proliferation, Migration, and Epithelial-Mesenchymal Transition of Human Lens Epithelial Cells by the lncRNA-MALAT1/miR-26a-5p/TET1 Signaling Axis 