Inhibitory Effect on Cerebral Inflammatory Response following Traumatic Brain Injury in Rats: A Potential Neuroprotective Mechanism of N-Acetylcysteine
Figure 5
Alterations in brain water content in SHAM group (), TBI group
(n = 6), and TBI-NAC group (n = 6). The brain water
content of the injured side was increased significantly at 3 days after TBI.
NAC treatment markedly reduced brain water content. No difference of brain
water content was detected in the uninjured side among the three groups. versus SHAM group; versus TBI group.