Mitochondrial Dysfunction: A Basic Mechanism in Inflammation-Related Non-Communicable Diseases and Therapeutic Opportunities
Figure 5
Mitochondrial fusion (a) and fission (b) processes in the liver (arrows). Mitochondrial morphology is basically controlled by metabolism and inflammation, and each change in morphology is mediated by large guanosine triphosphatases of the dynamin family, consistent with a model in which the capacity for oxidative phosphorylation is maximised under stressful conditions.
