Glutamine Administration Attenuates Kidney Inflammation in Obese Mice Complicated with Polymicrobial Sepsis

Table 3

Plasma concentrations of kidney function marker and inflammatory chemokine among groups.

	NC	SS12h	SG12h	SS24h	SG24h	SS48h	SG48h
NGAL (μg/dL)					^#
BUN (mg/dL)
Cre (mg/dL)
KC (ng/mL)			^#
MCP-1 (ng/mL)

Data are presented as the . NC: normal control group; SS: sepsis group with saline injection sacrificed at 12, 24, and 48 h after cecal ligation and puncture (CLP); SG: sepsis group with glutamine injection sacrificed at 12, 24, and 48 h after CLP; NGAL: neutrophil gelatinase-associated lipocalin-2; BUN: blood urea nitrogen; Cre: creatinine; KC: keratinocyte-derived chemokine; MCP-1: monocyte chemoattractant protein. Differences between 2 sepsis groups at the same time point were analyzed by -test. The comparison among NC and the sepsis groups at three different time points were analyzed by a one-way analysis of variance (ANOVA) followed by Tukey’s post hoc test. Significantly differs from other sepsis groups; ^#significantly differs from the SS group at the same time point ().