Lipopolysaccharide Inhibits Autophagy and Promotes Inflammatory Responses via p38 MAPK-Induced Proteasomal Degradation of Atg13 in Hepatic Stellate Cells

<div>Immunohistochemical staining were conducted in liver tissues from patients with ACLF. The tissues from ACLF patients were firstly stained with hematoxylin-eosin (HE) (a, b), then the expression of <i>α</i>-SMA (c, d) and p38 MAPK (e, f). Original magnification 100x (a, c, and e) or 200x (b, d, and f).</div>

Mediators of Inflammation

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Figure 2: Lipopolysaccharide Inhibits Autophagy and Promotes Inflammatory Responses via p38 MAPK-Induced Proteasomal Degradation of Atg13 in Hepatic Stellate Cells 

Figure 2 | Lipopolysaccharide Inhibits Autophagy and Promotes Inflammatory Responses via p38 MAPK-Induced Proteasomal Degradation of Atg13 in Hepatic Stellate Cells 