Therapeutic Potentials of Synapses after Traumatic Brain Injury: A Comprehensive Review

Figure 1

A brief drawing of the synaptic interaction with constant structural, metabolic, neuroendocrine, and inflammatory mechanisms after TBI; AMPAR: α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor; NMDAR: N-methyl-D-aspartate receptor; Glu: glutamate.