Probucol Attenuates Oxidative Stress, Energy Starvation, and Nitric Acid Production Following Transient Forebrain Ischemia in the Rat Hippocampus
Figure 1
Effects of forebrain ischemia, probucol and their combination on the number of intact neurons in CA1 subregion of rat hippocampus. Hippocampal CA1 subregion from the control (a), sham-operated (b), and probucol-treated (d) groups showing normal intact neurons (×20). Hippocampal CA1 subregion from rats subjected to 10 min forebrain ischemia group (c) showed severe and marked decrease in the number of intact neurons (×20). Hippocampal CA1 subregion from rats treated with probucol plus ischemia group (e) showed normal intact neurons (×20).
