Intensification of Doxorubicin-Related Oxidative Stress in the Heart by Hypothyroidism Is Not Related to the Expression of Cytochrome P450 NADPH-Reductase and Inducible Nitric Oxide Synthase, As Well As Activity of Xanthine Oxidase

<table>Representative mRNA iNOS content in cardiac muscle. HPRT was used as endogenous control. The results express percent changes (mean ±SD) with respect to the control group, which has been set at 100%. *<i>P </i>&lt; 0.05 versus control.</table>

Oxidative Medicine and Cellular Longevity

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Figure 4

Figure 4: Intensification of Doxorubicin-Related Oxidative Stress in the Heart by Hypothyroidism Is Not Related to the Expression of Cytochrome P450 NADPH-Reductase and Inducible Nitric Oxide Synthase, As Well As Activity of Xanthine Oxidase 

Figure 4 | Intensification of Doxorubicin-Related Oxidative Stress in the Heart by Hypothyroidism Is Not Related to the Expression of Cytochrome P450 NADPH-Reductase and Inducible Nitric Oxide Synthase, As Well As Activity of Xanthine Oxidase 