Increased Clearance of Reactive Aldehydes and Damaged Proteins in Hypertension-Induced Compensated Cardiac Hypertrophy: Impact of Exercise Training

<div>Cardiac levels of small chaperones and apoptosis-related markers are unchanged in compensated hypertrophy in hypertensive rats. Protein levels of small chaperones <i>αβ</i>-crystallin (a) and HSP25 (b), and apoptosis-related markers Bad (c), phospho Bad at serine 112 (d) and Bcl-2 (e), and their representative blots (f) in heart lysate from control (normotensive), sedentary spontaneously hypertensive rats (SHR), and swimming-trained SHR (SHRt). Protein expression was normalized by GAPDH. Data are presented as mean ± SEM.</div>

Oxidative Medicine and Cellular Longevity

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Figure 5

Figure 5: Increased Clearance of Reactive Aldehydes and Damaged Proteins in Hypertension-Induced Compensated Cardiac Hypertrophy: Impact of Exercise Training 

Figure 5 | Increased Clearance of Reactive Aldehydes and Damaged Proteins in Hypertension-Induced Compensated Cardiac Hypertrophy: Impact of Exercise Training 