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Oxidative Medicine and Cellular Longevity
Volume 2016, Article ID 9648769, 9 pages
http://dx.doi.org/10.1155/2016/9648769
Research Article

Nr2e1 Deficiency Augments Palmitate-Induced Oxidative Stress in Beta Cells

Department of Endocrinology, Zhongnan Hospital of Wuhan University, Wuhan, Hubei 430071, China

Received 13 April 2015; Revised 12 July 2015; Accepted 15 July 2015

Academic Editor: Ada Popolo

Copyright © 2016 Xiaoli Shi et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Citations to this Article [4 citations]

The following is the list of published articles that have cited the current article.

  • Jingqi Fu, Qi Cui, Bei Yang, Yongyong Hou, Huihui Wang, Yuanyuan Xu, Difei Wang, Qiang Zhang, and Jingbo Pi, “The impairment of glucose-stimulated insulin secretion in pancreatic β-cells caused by prolonged glucotoxicity and lipotoxicity is associated with elevated adaptive antioxidant response,” Food and Chemical Toxicology, 2016. View at Publisher · View at Google Scholar
  • George Kefalas, and Louise Larose, “PERK leads a hub dictating pancreatic β cell homoeostasis,” Biology of the Cell, 2017. View at Publisher · View at Google Scholar
  • Jingjing Wang, and Hongjun Wang, “Oxidative Stress in Pancreatic Beta Cell Regeneration,” Oxidative Medicine and Cellular Longevity, vol. 2017, pp. 1–9, 2017. View at Publisher · View at Google Scholar
  • G. Sun, Q. Cui, and Y. Shi, “Nuclear Receptor TLX in Development and Diseases,” 2017. View at Publisher · View at Google Scholar