Research Article
Roles of Endoplasmic Reticulum Stress in NECA-Induced Cardioprotection against Ischemia/Reperfusion Injury
Figure 3
The effect of NECA on GSK-3β at Ser9 phosphorylation and GRP94 protein expression in H2O2-treated H9c2 cells. (a) H2O2 (800 μM) increased GSK-3β phosphorylation and decreased GRP94 expression, the effect that was blocked by NECA (0.1 μM). (b) Data are mean ± SD for 8 independent experiments performed in duplicate. compared to the control group; compared to NECA.
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