Effects of Glutathione S-Transferase Gene Polymorphisms and Antioxidant Capacity per Unit Albumin on the Pathogenesis of Chronic Obstructive Pulmonary Disease

<table class="table-group" id="tab1"><tr><td><table class="table"><tr><td class="thead-hr" colspan="2"><hr/></td></tr><tr class="thead"><td class="align_left">Symptoms or parameters</td><td class="align_center">Values</td></tr><tr><td class="thead-hr" colspan="2"><hr/></td></tr><tr><td class="align_left">Cough</td><td class="align_center">100 (%)</td></tr><tr><td class="align_left">Expectoration</td><td class="align_center">100 (%)</td></tr><tr><td class="align_left">Dyspnea</td><td class="align_center">79 (%)</td></tr><tr><td class="align_left" colspan="2"><hr/></td></tr><tr><td class="align_left">FEV1/FVC</td><td class="align_center">0.60 ± 0.06 (mean ± SD)</td></tr><tr><td class="align_left">FEV1% estimated value</td><td class="align_center">69.56 ± 7.91 (mean ± SD)</td></tr><tr class="table-tr"><td colspan="2"><hr class="tbody-hr"/></td></tr></table></td></tr></table>

The clinical characteristics and spirometric parameters of COPD patients.

Oxidative Medicine and Cellular Longevity

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Table 1

Table 1: Effects of Glutathione S-Transferase Gene Polymorphisms and Antioxidant Capacity per Unit Albumin on the Pathogenesis of Chronic Obstructive Pulmonary Disease 

Table 1 | Effects of Glutathione S-Transferase Gene Polymorphisms and Antioxidant Capacity per Unit Albumin on the Pathogenesis of Chronic Obstructive Pulmonary Disease 