Research Article

Inhibition of miR-302 Suppresses Hypoxia-Reoxygenation-Induced H9c2 Cardiomyocyte Death by Regulating Mcl-1 Expression

Figure 6

H9c2 cardiomyocytes subjected to H/R injury in the presence of different concentrations of nonsense control (NC), miR-302 mimic (m302), and/or miR-302 antagomir (i302). (a) Activated caspase-3, (b) activated caspase-9, and (c) cleaved PARP in different groups were analyzed by western blot. Β-actin was used as an internal control. Data are presented as the mean ± STD of six analyses. between two groups.
(a) Activated caspase-3 expression
(b) Activated caspase-9 expression
(c) Cleaved PARP expression