Evaluation of Oxidative Stress in Cardiomyocytes during the Aging Process in Rats Treated with Resveratrol

Table 1

Main cardioprotective activities of resveratrol.

Activity	Concentration	Reference
Reduction of cardiovascular structural and functional deterioration in CHF.	5.0 mg/kg	Ahmet et al. [36]
Attenuation of postinfarct cardiac remodeling and contractile dysfunction.	2.5 mg/kg	Raj et al. [37]
Activation of a novel deacetylating pathway and attenuation of cardiac oxidative stress in diabetic heart.	10.0 mg/kg	Bagul et al. [38]
Induces autophagy and protects hearts from doxorubicin-induced toxicity.	5.0–50.0 mg/kg	Dutta et al. [39]
Prevents oxidative stress induced cardiomyocyte injury mainly by preserving the activities of critical antioxidant enzymes.	2.5 mg/kg	Movahed et al. [40]
Increase of expression of AK1 and IDPm on ventricular modeling.	1.0 mg/kg	Lin et al. [41]
Reverse of abnormalities in diastolic heart function associated with high-fat feeding in obese prone rats.	2.5 mg/kg	Louis et al. [42]
Suppression of sympathetic neural remodeling process after myocardial infarction.	1.0 mg/kg	Xin et al. [43]
Beneficial effects on myocardial function, coronary perfusion, EC function, and vascular tone.	10.0 mg/kg	Joshi et al. [44]
Protection against recurrent stroke.	25.0 mg/kg	Clark et al., [45]