Research Article
Ex Vivo Cardiotoxicity of Antineoplastic Casiopeinas Is Mediated through Energetic Dysfunction and Triggered Mitochondrial-Dependent Apoptosis
Figure 5
Effect of Cas on isolated heart on TBARS content (a), aconitase activity (b), and ANT/thiols groups (c) on mitochondria from IC50 Cas-treated and Doxo (5 μM) hearts. Values are mean ± SEM. versus control ( animals for each treatment, except for Doxo group ()).
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