Fisetin Confers Cardioprotection against Myocardial Ischemia Reperfusion Injury by Suppressing Mitochondrial Oxidative Stress and Mitochondrial Dysfunction and Inhibiting Glycogen Synthase Kinase 3<i>β</i> Activity

<div>This scheme shows the fisetin cardioprotective effects against myocardial tissue injury upon I/R. Fisetin scavenges ROS and prevents mitochondrial dysfunction by inducing the Nrf-1/2 expression, bolstering the mitochondrial physiology, and inhibiting the GSK3<i>β</i> activity.</div>

Oxidative Medicine and Cellular Longevity

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Figure 10

Figure 10: Fisetin Confers Cardioprotection against Myocardial Ischemia Reperfusion Injury by Suppressing Mitochondrial Oxidative Stress and Mitochondrial Dysfunction and Inhibiting Glycogen Synthase Kinase 3<i>β</i> Activity 

Figure 10 | Fisetin Confers Cardioprotection against Myocardial Ischemia Reperfusion Injury by Suppressing Mitochondrial Oxidative Stress and Mitochondrial Dysfunction and Inhibiting Glycogen Synthase Kinase 3β Activity 