Overexpression of miR-27b-3p Targeting Wnt3a Regulates the Signaling Pathway of Wnt/<i>β</i>-Catenin and Attenuates Atrial Fibrosis in Rats with Atrial Fibrillation

<div>The molecular mechanism diagram showed that miR-27b-3p played an important role in atrial fibrosis and the occurrence of AF by regulation of the signaling pathway of Wnt/<i>β</i>-Catenin through targeted Wnt3a.</div>

Oxidative Medicine and Cellular Longevity

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Figure 10

Figure 10: Overexpression of miR-27b-3p Targeting Wnt3a Regulates the Signaling Pathway of Wnt/<i>β</i>-Catenin and Attenuates Atrial Fibrosis in Rats with Atrial Fibrillation 

Figure 10 | Overexpression of miR-27b-3p Targeting Wnt3a Regulates the Signaling Pathway of Wnt/β-Catenin and Attenuates Atrial Fibrosis in Rats with Atrial Fibrillation 