N-Acetyltransferase 10 Enhances Doxorubicin Resistance in Human Hepatocellular Carcinoma Cell Lines by Promoting the Epithelial-to-Mesenchymal Transition

<div>Knocking down and inhibiting NAT10 increase the chemosensitivity of HCC cell lines to doxorubicin. (a) Western blotting confirmation of NAT10 knockdown efficiency in siRNA-transfected cells. (b–e) CCK-8 assay of the viability of HCC cells treated with doxorubicin, the NAT10 siRNA, or doxorubicin and remodelin following knockdown of NAT10.</div>

Oxidative Medicine and Cellular Longevity

fig2

Figure 2

Figure 2: N-Acetyltransferase 10 Enhances Doxorubicin Resistance in Human Hepatocellular Carcinoma Cell Lines by Promoting the Epithelial-to-Mesenchymal Transition 

Figure 2 | N-Acetyltransferase 10 Enhances Doxorubicin Resistance in Human Hepatocellular Carcinoma Cell Lines by Promoting the Epithelial-to-Mesenchymal Transition 