N-Acetyltransferase 10 Enhances Doxorubicin Resistance in Human Hepatocellular Carcinoma Cell Lines by Promoting the Epithelial-to-Mesenchymal Transition

<div>Inhibition of NAT10 reverses the doxorubicin-induced EMT in HCC cell lines. Western blotting and immunofluorescence analysis of the expression of EMT-related markers E-cadherin and vimentin in cells treated with doxorubicin or remodelin (a, b) or HCC cells transfected with the NAT10 siRNA (c).</div>

Oxidative Medicine and Cellular Longevity

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Figure 4: N-Acetyltransferase 10 Enhances Doxorubicin Resistance in Human Hepatocellular Carcinoma Cell Lines by Promoting the Epithelial-to-Mesenchymal Transition 

Figure 4 | N-Acetyltransferase 10 Enhances Doxorubicin Resistance in Human Hepatocellular Carcinoma Cell Lines by Promoting the Epithelial-to-Mesenchymal Transition 