Melatonin Ameliorates MI-Induced Cardiac Remodeling and Apoptosis through a JNK/p53-Dependent Mechanism in Diabetes Mellitus

<div>Schematic diagram depicting the proposed signaling pathways involving JNK, p53, and apoptosis behind the melatonin-offered protective effect against post-MI injury in diabetes.</div>

Oxidative Medicine and Cellular Longevity

fig8

Figure 8

Figure 8: Melatonin Ameliorates MI-Induced Cardiac Remodeling and Apoptosis through a JNK/p53-Dependent Mechanism in Diabetes Mellitus 