Corrigendum to “Salvianolic Acid A Inhibits OX-LDL Effects on Exacerbating Choroidal Neovascularization via Downregulating CYLD”

<div>Sal A modulates CYLD via PI3K/AKT/mTOR pathway. (a) As in our former study [<a href="/journals/omcl/2020/1769871/#B2">2</a>], Sal A promoted ERK and PI3K/AKT/mTOR phosphorylation in ARPE-19 cells after 48 hours. From left to right: control (C), oxidized low-density lipoprotein (OX), OX-LDL plus 5 <i>μ</i>M salvianolic acid A (OX-LDL 5), and OX-LDL plus 50 <i>μ</i>M salvianolic acid A (OX-LDL 50), all for 24 hours; C, OX, OX-LDL 5, and OX-LDL 50, all for 48 hours.</div>

Oxidative Medicine and Cellular Longevity

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Figure 10

Figure 10: Corrigendum to “Salvianolic Acid A Inhibits OX-LDL Effects on Exacerbating Choroidal Neovascularization via Downregulating CYLD” 