Research Article
Peroxiredoxin-1 Overexpression Attenuates Doxorubicin-Induced Cardiotoxicity by Inhibiting Oxidative Stress and Cardiomyocyte Apoptosis
Figure 3
Prdx1 overexpression attenuated cardiac dysfunction induced by DOX. (a, b) Echocardiographic parameters for each group (). (c–g) Hemodynamic parameters for each group (). compared with the NS group; # compared with the DOX group.
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