Pathophysiologic Mechanisms of Cardiovascular Disease in Obstructive Sleep Apnea Syndrome

Table 1

Prevalence studies on obstructive sleep apnea syndrome.

Study	Population	Age	Method	Criteria	SDB prevalence	OSAS
Durán et al. 2001 [14]	2148 subjects from the electoral census	30–70	(1) Questionnaire; (2) Validated portable instrument in 442 subjects; (3) PSG in 555 subjects	AHI ≥ 5	26.3% (M) and 28% (F)
				AHI ≥ 10	19% (M) and 14.9% (F)
				AHI ≥ 15	14.2% (M) and 8.6% (F)
				AHI ≥ 20	9.6% (M) and 6% (F)
				AHI ≥ 30	6.8% (M) and 4.3% (F)
				OSAS = AHI ≥ 10 plus Symptoms		3.4% M; 3% F
Udwadia et al., 2004 [15]	658 healthy urban Indian subjects	35–65	(1) Questionnaire; (2) PSG on subgroup in 250 subjects	AHI ≥ 5	19.5%	7.5%
				AHI ≥ 10	11.1%	6.1%
				AHI ≥ 15	8.4%	5.4%
				OSAS = AHI plus Symptoms
Sharma et al., 2006 [16]	2150 semiurban community in Delhi	30–60	(1) Questionnaire; (2) PSG on subgroup in 150 subjects	AHI ≥ 5	13.7%
				OSAS = AHI ≥ 5 plus Symptoms		3.6%
Pływaczewski et al., 2008 [17]	1503 from Warsaw electoral registers	Over 30 years of age	(1) Questionnaire; (2) PSG on subgroup in 676 subjects	AHI ≥ 10	14.3%
				OSAS = AHI ≥ 10 plus Symptoms		7.5%

Abbreviations: AHI: apnea hypopnea index; PSG: polysomnography; SDB: sleep disordered breathing; M: male; F: female.