Regulation of Glial Cell Functions by PPAR-<svg style="vertical-align:-3.636pt;width:12.3px;" id="M1" height="15.0125" version="1.1" viewBox="0 0 12.3 15.0125" width="12.3"  xmlns="http://www.w3.org/2000/svg">
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<table>Cellular targets of PPAR-<i>γ</i> agonists in neurodegenerative diseases. PPAR-<i>γ</i>
agonists can control neuroinflammation, neurodegeneration, and demyelination by
effecting several cellular targets and by several direct and indirect
mechanisms. PPAR-<i>γ</i> agonists can control glial activation, preventing a number of proinflammatory
activities that can contribute to myelin/OL damage and neurotoxicity PPAR-<i>γ</i>
agonists may also affect OLs and neurons, by preventing release inflammatory
mediators and/or promote the synthesis of soluble factors or membrane-bound
molecules that control glial activation.</table>

PPAR Research

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Figure 1: Regulation of Glial Cell Functions by PPAR-<svg style="vertical-align:-3.636pt;width:12.3px;" id="M1" height="15.0125" version="1.1" viewBox="0 0 12.3 15.0125" width="12.3"  xmlns="http://www.w3.org/2000/svg">
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PPAR Research

Regulation of Glial Cell Functions by PPAR- 𝜸 Natural and Synthetic Agonists

Figure 1

Regulation of Glial Cell Functions by PPAR- Natural and Synthetic Agonists