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PPAR Research
Volume 2011, Article ID 318134, 11 pages
http://dx.doi.org/10.1155/2011/318134
Research Article

Electrophilic PPARγ Ligands Attenuate IL-1β and Silica-Induced Inflammatory Mediator Production in Human Lung Fibroblasts via a PPARγ-Independent Mechanism

1Division of Pulmonary and Critical Care Medicine, University of Rochester, 601 Elmwood Avenue, P.O. Box 692, Rochester, NY 14642, USA
2Department of Environmental Medicine, University of Rochester, Rochester, NY 14642, USA
3Lung Biology and Disease Program, University of Rochester, Rochester, NY 14642, USA
4Department of Surgery, University of Rochester, Rochester, NY 14642, USA

Received 23 November 2010; Revised 4 March 2011; Accepted 25 March 2011

Academic Editor: Paul Drew

Copyright © 2011 Christopher M. Hogan et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

How to Cite this Article

Christopher M. Hogan, Thomas H. Thatcher, Ramil E. Sapinoro, et al., “Electrophilic PPAR Ligands Attenuate IL-1 and Silica-Induced Inflammatory Mediator Production in Human Lung Fibroblasts via a PPAR -Independent Mechanism,” PPAR Research, vol. 2011, Article ID 318134, 11 pages, 2011. https://doi.org/10.1155/2011/318134.