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PPAR Research
Volume 2016, Article ID 3082340, 7 pages
Review Article

PPARδ as a Metabolic Initiator of Mammary Neoplasia and Immune Tolerance

Department of Oncology, Georgetown University Medical Center and the Lombardi Comprehensive Cancer Center, 3970 Reservoir Rd, NW, Washington, DC 20007, USA

Received 19 July 2016; Accepted 3 November 2016

Academic Editor: Stefano Caruso

Copyright © 2016 Robert I. Glazer. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Citations to this Article [3 citations]

The following is the list of published articles that have cited the current article.

  • Robert I. Glazer, and Levy Kopelovich, “PPARs as determinants of the estrogen receptor lineage: Use of synthetic lethality for the treatment of estrogen receptornegative breast cancer,” Oncotarget, vol. 8, no. 31, pp. 50337–50341, 2017. View at Publisher · View at Google Scholar
  • David J. Schultz, Abirami Krishna, Stephany L. Vittitow, Negin Alizadeh-Rad, Penn Muluhngwi, Eric C. Rouchka, and Carolyn M. Klinge, “Transcriptomic response of breast cancer cells to anacardic acid,” Scientific Reports, vol. 8, no. 1, 2018. View at Publisher · View at Google Scholar
  • Shreekrishna Lamichane, Babita Dahal Lamichane, and Sang-Mo Kwon, “Pivotal Roles of Peroxisome Proliferator-Activated Receptors (PPARs) and Their Signal Cascade for Cellular and Whole-Body Energy Homeostasis,” International Journal of Molecular Sciences, vol. 19, no. 4, pp. 949, 2018. View at Publisher · View at Google Scholar