Alteration in Marrow Stromal Microenvironment and Apoptosis Mechanisms Involved in Aplastic Anemia: An Animal Model to Study the Possible Disease Pathology
Figure 4
Colony forming efficiency of the bone marrow-derived cells from the normal and aplastic anemic groups of mice. (a) Colony forming unit erythrocytes (CFU-E) from the normal and aplastic bone marrow cells. (b) Erythroid burst forming unit (BFU-E) in normal and AA. (c) Colony forming unit granulocytes (CFU-G) by the AA and normal bone marrow cells. (d) Colony forming unit granulocytes macrophages (CFU-GM) from the AA and normal bone marrow cells. (e) Colony forming unit granulocyte erythrocyte monocyte macrophage (CFU-GEMM) in normal and AA. (f) Colony forming unit fibroblasts form the normal and AA bone marrow cells. All the results represent the relative number of CFUs in aplastic anemia as compared to the normal and correspond to the median ± S.D. levels observed from 15 () samples studied ().
