Research Article
IGF1 and NRG1 Enhance Proliferation, Metabolic Maturity, and the Force-Frequency Response in hESC-Derived Engineered Cardiac Tissues
Figure 6
NRG1 treatment increases mitochondrial activity in cardiomyocytes. Basal metabolic profiling of hESC-cardiomyocytes was normalized to live cells (with LIVE/DEAD® staining) to determine oxygen consumption rate (OCR) (a), extracellular acidification rate (ECAR), and the OCR/ECAR ratio (b) (). Data are expressed as mean ± SD. and versus control.
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