Research Article

Quality of Chronic Anticoagulation Control in Patients with Intracranial Haemorrhage due to Vitamin K Antagonists

Table 3

Treatment.

TREATMENT

Haemorrhage treatment N (%)
(i) None16 (30.8%)
(ii) Vitamin K6 (11.5%)
(iii) Prothrombin0 (0%)
(iv) Prothrombin and vitamin K26 (50%)
(v) Surgical4 (7.7%)

Treatment at discharge N (%)
(i) None10 (41.6%)
(ii) Antiplatelet agents3 (12.5%)
(iii) VKAs2 (8.3%)
(iv) VKAs and antiplatelet agents0 (0%)
(v) Dabigatran2 (8.3%)
(vi) Rivaroxaban0 (0%)
(vii) Apixaban0 (0%)
(viii) NOAC and aspirin0 (0%)
(ix) LAA closure5 (20.8)
(x) Prophylactic heparin

Treatment at 3 months N (%)
(i) None0 (0%)
(ii) Antiplatelet agents2 (9.1%)
(iii) VKAs2 (9.1%)
(iv) VKAs and antiplatelet agents0 (0%)
(v) Dabigatran4 (18.2%)
(vi) Rivaroxaban7 (31.8 %)
(vii) Apixaban2 (9.1%)
(viii) NOAC and aspirin0 (0%)
(ix) LAA closure3 (13.6%)
(x) Prophylactic heparin1 (4.5%)

Treatment changed 3 months after discharge.15/22

VKA: Vitamin K antagonist. NOAC: novel oral anticoagulant. LAA: left atrial appendage.