NMDA Receptor-Mediated Neuroprotective Effect of the <i>Scutellaria baicalensis</i> Georgi Extract on the Excitotoxic Neuronal Cell Death in Primary Rat Cortical Cell Cultures

<table>Inhibition of N-methyl-D-aspartate (NMDA) or glutamate- (Glu-) induced excitotoxicity by the ethanol extract of<i> Scutellaria baicalensis</i> (S.B.). Lactate dehydrogenase (LDH) activities given off by the damaged neurons into the culture media were measured at 20–24 h after the exposure. Data were calculated as percent of control LDH activity released into the Glu-treated culture medium. Cultures were exposed for 20 min to 350 mM Glu or NMDA in the presence of various concentrations (1, 10, 25, 50, and 100 <i>μ</i>g/mL) of the AGR extract and maintained as described in the experimental part.</table>

The Scientific World Journal

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Figure 2: NMDA Receptor-Mediated Neuroprotective Effect of the <i>Scutellaria baicalensis</i> Georgi Extract on the Excitotoxic Neuronal Cell Death in Primary Rat Cortical Cell Cultures 

Figure 2 | NMDA Receptor-Mediated Neuroprotective Effect of the Scutellaria baicalensis Georgi Extract on the Excitotoxic Neuronal Cell Death in Primary Rat Cortical Cell Cultures 