[Retracted] Targeting Mitochondria as Therapeutic Strategy for Metabolic Disorders

<table>Mitochondrial dysfunction and hypoxia in cancer. Schematic representation of the role of mitochondrial dysfunction in tumorigenesis. Damaged mitochondria release ROS which on turn activates HIF1<i>α</i>. Finally, HIF1<i>α</i> activates the tumorigenic hypoxia pathway by initiating transcription of genes involved in glucose metabolism, angiogenesis, and survival.</table>

The Scientific World Journal

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Figure 2

Figure 2: [Retracted] Targeting Mitochondria as Therapeutic Strategy for Metabolic Disorders 