Research Article

Potassium Concentration on Admission Is an Independent Risk Factor for Target Lesion Revascularization in Acute Myocardial Infarction

Table 2

Clinical and angiographic characteristics of acute myocardial infarction.

Low K (<3.9)Mid K (≥3.9, <4.3)High K (≥4.3)P value

Time from chest pain to door (hr)
SBP on admission (mmHg)
HR on admission (mmHg)
Previous MI4 (4.35)7 (7.61)9 (9.78)0.359
Preinfarction angina36 (39.1)36 (39.1)25 (27.2)0.146
STEMI73 (79.3)70 (76.0)61 (66.3)0.111
Anterior MI50 (54.3)42 (45.7)41 (44.6)0.347
Killip > I17 (18.5)13 (14.1)27 (29.3)0.0318
Coronary multivessel involvement (%)35 (38.0)33 (35.9)36 (39.1)0.898
PG on admission (mg/dL)
WBC count on admission (/mm3)
Creatinine on admission (mg/dL)
Hemoglobin on admission (g/dL)
Potassium on admission (mEq/L)
Stent diameter implanted in culprit lesion (mm)
Stent length implanted in culprit lesion (mm)
TIMI grade 3 after PCI76 (82.6)73 (79.3)74 (80.4)0.849
Peak CK

SBP: systolic blood pressure; HR: heart rate; MI: myocardial infarction; STEMI: ST-elevation myocardial infarction; PG: plasma glucose; WBC: white blood cell; TIMI: thrombolysis in myocardial infarction; PCI: percutaneous coronary intervention; CK: creatine phosphokinase. P < 0.05 versus low K; P < 0.05 versus mid K.