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Oxidative Medicine and Cellular Longevity
Volume 2017 (2017), Article ID 1459497, 8 pages
Review Article

The Beneficial Effects of Quercetin, Curcumin, and Resveratrol in Obesity

1Laboratory of Molecular Pharmacology, Department of Pharmacology, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China
2Experiment Center for Medical Science Research, Kunming Medical University, Kunming, Yunnan, China
3Department of Hematology and Oncology, The Children’s Hospital of Soochow, Jiangsu, China
4Department of Gastroenterology, The Third Affiliated Hospital of Soochow University, Changzhou, Jiangsu, China
5School of Chinese Medicine, Hong Kong Baptist University, Kowloon, Hong Kong
6Key Laboratory of Medical Electrophysiology of Ministry of Education, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China

Correspondence should be addressed to Zhangang Xiao

Received 7 April 2017; Revised 20 July 2017; Accepted 2 August 2017; Published 24 August 2017

Academic Editor: Lin-sen Qing

Copyright © 2017 Yueshui Zhao et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.


Over the past two decades, obesity has been one of the major public health concerns in most countries. In the search for new molecules that could be used for the treatment of obesity, good perspectives have been opened up for polyphenols, a class of natural bioactive phytochemicals. Experimental and limited clinical trial evidence supports that some polyphenols such as quercetin, curcumin, and resveratrol have potential benefit functions on obesity treatment. This brief review focuses on the main functions of the above-named polyphenols on adipose tissue. These polyphenols may play beneficial effects on adipose tissue under obese condition by alleviating intracellular oxidative stress, reducing chronic low-grade inflammation, inhibiting adipogenesis and lipogenesis, and suppressing the differentiation of preadipocytes to mature adipocytes.