BioMed Research International

Inflammatory Reaction in Neurological Diseases


Publishing date
25 Jul 2014
Status
Published
Submission deadline
07 Mar 2014

Lead Editor

1Department of Neurosurgery, Kaohsiung Chang Gung Memorial Hospital, Chang Gung University, College of Medicine, Kaohsiung, Taiwan

2Cheng-Hsien Lu, Department of Neurology, Kaohsiung Chang Gung Memorial Hospital, Kaohsiung, Taiwan; Department of Biological Science, National Sun Yat-Sen University, Kaohsiung, Taiwan

3Department of Anesthesiology, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan

4Department of Neuropathology, University of Pittsburgh Medical Center, Pittsburgh, PA, USA


Inflammatory Reaction in Neurological Diseases

Description

Inflammatory reaction in the central nervous system (CNS) is now recognized to be a feature of all neurological disorders. In neurological degenerative diseases, such as multiple sclerosis, Parkinson’s disease, Alzheimer’s disease, and chronic neuropathic pain, there is prominent infiltration of various leukocyte subsets into the CNS or there is intense activation of microglia with resultant elevation of many inflammatory mediators within the CNS. For acute critical CNS diseases, such as delayed deterioration associated with vasospasm after subarachnoid hemorrhage (SAH), ischemic stroke, spontaneous intracerebral hemorrhage (ICH), and traumatic brain injury (TBI), recent further evidence shows that inflammation may be a potential target for therapy. Inflammation has become a promising area of research for new treatments. Knowledge and understanding of these conditions have led to the development of animal models, successful therapies, and novel tools to characterize these clinical conditions and provide better care to patients.

We invite authors to submit original research and review articles that will stimulate the continuing efforts to understand the molecular pathology underlying inflammatory neurological disease, the development of strategies to treat these conditions, and the evaluation of outcomes. We are particularly interested in articles describing the new modalities for clinical characterization of inflammatory neurological disease and measuring outcomes from treatment trials, advances in molecular diagnostics, new insights into inflammatory neurological disease using animal models, and current concepts in the treatment of inflammatory neurological disease. Potential topics include, but are not limited to:

  • Recent developments in inflammatory neurological disease research, especially on ischemic/hemorrhagic stroke patients
  • Identifying potential biomarkers/correlates of inflammation to predictive stroke in patients with immunocompromised
  • Latest technologies for clinical evaluation and measuring outcomes, especially neural inflammation in neuroimage findings
  • New cellular and animal models to test and understand inflammatory neurological disease
  • Recent advances in CNS drug delivery

Before submission authors should carefully read over the journal’s Author Guidelines, which are located at http://www.hindawi.com/journals/bmri/guidelines/. Prospective authors should submit an electronic copy of their complete manuscript through the journal Manuscript Tracking System at http://mts.hindawi.com/submit/journals/bmri/neuroscience/irnd/ according to the following timetable:


Articles

  • Special Issue
  • - Volume 2014
  • - Article ID 408514
  • - Editorial

Inflammatory Reaction in Neurological Diseases

Hung-Chen Wang | Cheng-Hsien Lu | ... | Jason Cheng-Hsuan Chiang
  • Special Issue
  • - Volume 2014
  • - Article ID 309129
  • - Review Article

Chronic Neuroinflammation in Alzheimer’s Disease: New Perspectives on Animal Models and Promising Candidate Drugs

Christopher Millington | Sandra Sonego | ... | Gerald Münch
  • Special Issue
  • - Volume 2014
  • - Article ID 910260
  • - Research Article

Simvastatin Combined with Antioxidant Attenuates the Cerebral Vascular Endothelial Inflammatory Response in a Rat Traumatic Brain Injury

Kuo-Wei Wang | Hao-Kuang Wang | ... | Kang Lu
  • Special Issue
  • - Volume 2014
  • - Article ID 723176
  • - Research Article

Correlation of Altered Expression of the Autophagy Marker LC3B with Poor Prognosis in Astrocytoma

Daniel Winardi | Hung-Pei Tsai | ... | Ching-Liang Hsieh
  • Special Issue
  • - Volume 2014
  • - Article ID 256879
  • - Research Article

Association between Oxidative Stress and Outcome in Different Subtypes of Acute Ischemic Stroke

Nai-Wen Tsai | Ya-Ting Chang | ... | Cheng-Hsien Lu
  • Special Issue
  • - Volume 2014
  • - Article ID 598762
  • - Research Article

Elevated Serum Vascular Cell Adhesion Molecule-1 Is Associated with Septic Encephalopathy in Adult Community-Onset Severe Sepsis Patients

Chih-Min Su | Hsien-Hung Cheng | ... | Cheng-Hsien Lu
  • Special Issue
  • - Volume 2014
  • - Article ID 861812
  • - Research Article

Association between Autoimmune Rheumatic Diseases and the Risk of Dementia

Kang Lu | Hao-Kuang Wang | ... | Kuen-Jer Tsai
  • Special Issue
  • - Volume 2014
  • - Article ID 129796
  • - Research Article

Predictors of Memory and Processing Speed Dysfunctions after Traumatic Brain Injury

William Winardi | Aij-Lie Kwan | ... | Chwen-Yng Su
  • Special Issue
  • - Volume 2014
  • - Article ID 506458
  • - Research Article

Differential Hypermethylation of Death-Associated Protein Kinase Promoter in Central Neurocytoma and Oligodendroglioma

Chia-Li Chung | Hung Pei Tsai | ... | Aij-Lie Kwan
  • Special Issue
  • - Volume 2014
  • - Article ID 275178
  • - Review Article

Immune Responses in Parkinson’s Disease: Interplay between Central and Peripheral Immune Systems

Xiaomin Su | Howard J. Federoff
BioMed Research International
 Journal metrics
See full report
Acceptance rate8%
Submission to final decision110 days
Acceptance to publication24 days
CiteScore5.300
Journal Citation Indicator-
Impact Factor-
 Submit Check your manuscript for errors before submitting

Article of the Year Award: Impactful research contributions of 2022, as selected by our Chief Editors. Discover the winning articles.